This site uses cookies to measure how you use the website so it can be updated and improved based on your needs and also uses cookies to help remember the notifications you’ve seen, like this one, so that we don’t show them to you again. If you could also tell us a little bit about yourself, this information will help us understand how we can support you better and make this site even easier for you to use and navigate.

A randomized, double-blind, placebo-controlled trial of simvastatin to treat Alzheimer disease

Authors

Sano, M., Bell, K. L., Galasko, D., Galvin, J. E., Thomas, R. G., van Dyck, C. H., Aisen, P. S.

Journal

Neurology, Volume: 77, No.: 6, Pages.: 556-563

Year of Publication

2011

Abstract

Background: Lowering cholesterol is associated with reduced CNS amyloid deposition and increased dietary cholesterol increases amyloid accumulation in animal studies. Epidemiologic data suggest that use of 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (statins) may decrease the risk of Alzheimer disease (AD) and a single-site trial suggested possible benefit in cognition with statin treatment in AD, supporting the hypothesis that statin therapy is useful in the treatment of AD.; Objective: To determine if the lipid-lowering agent simvastatin slows the progression of symptoms in AD.; Methods: This randomized, double-blind, placebo-controlled trial of simvastatin was conducted in individuals with mild to moderate AD and normal lipid levels. Participants were randomly assigned to receive simvastatin, 20 mg/day, for 6 weeks then 40 mg per day for the remainder of 18 months or identical placebo. The primary outcome was the rate of change in the Alzheimer’s Disease Assessment Scale-cognitive portion (ADAS-Cog). Secondary outcomes measured clinical global change, cognition, function, and behavior.; Results: A total of 406 individuals were randomized: 204 to simvastatin and 202 to placebo. Simvastatin lowered lipid levels but had no effect on change in ADAS-Cog score or the secondary outcome measures. There was no evidence of increased adverse events with simvastatin treatment.; Conclusion: Simvastatin had no benefit on the progression of symptoms in individuals with mild to moderate AD despite significant lowering of cholesterol.; Classification Of Evidence: This study provides Class I evidence that simvastatin 40 mg/day does not slow decline on the ADAS-Cog.;

Bibtex Citation

@article{Sano_2011, doi = {10.1212/wnl.0b013e318228bf11}, url = {http://dx.doi.org/10.1212/WNL.0b013e318228bf11}, year = 2011, month = {jul}, publisher = {Ovid Technologies (Wolters Kluwer Health)}, volume = {77}, number = {6}, pages = {556--563}, author = {M. Sano and K. L. Bell and D. Galasko and J. E. Galvin and R. G. Thomas and C. H. van Dyck and P. S. Aisen}, title = {A randomized, double-blind, placebo-controlled trial of simvastatin to treat Alzheimer disease}, journal = {Neurology} }

Keywords

adverse effects, aged, alzheimer disease, apolipoproteins e, blood, cholesterol, cholesterol ldl, cholinesterase inhibitors, cognition, disease progression, double-blind method, drug therapy, female, genetics, humans, lipids, liver function tests, male, neuropsychological tests, nootropic agents, physiology, psychology, simvastatin, therapeutic use, treatment outcome

Countries of Study

USA

Types of Dementia

Alzheimer’s Disease

Types of Study

Randomised Controlled Trial

Type of Outcomes

ADLs/IADLs, Behaviour, Cognition

Type of Interventions

Pharmaceutical Interventions

Pharmaceutical Interventions

Other